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three operate by altering receptor expression for the plasma membrane, by facilitating improvements inside the ligand-binding area and consequent integrin activation, or by facilitating adjustments inside the conformation on the transmembrane and intracellular domains of α
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three to bind fibrinogen continually, but resulted in a serious bleeding syndrome and amplified murine mortality. J Thromb Haemost
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3, concomitant with increased pSrc associations in unstimulated platelets. These variations bring about altered Src and FAK phosphorylation concentrations, which come about without the need of noticed activation in the ligand-binding domain of α